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Bill Scott
Bill Scott is a co-investigator of a research project studying somatization disorders at UCLA’s Neuropsychiatric Institute with Lobsang Rapgay, Ph.D. He’s in the design process of a study investigating brain states during mindfulness meditation with Jeffery Schwartz, M.D. and Barry Sterman, Ph.D. He recently developed a new neuroimaging technology that creates a fractal signature from EEG and is successfully utilizing this technology as a form of EEG biofeedback in a process termed BrainPaint®. He is the first author and principal investigator of a controlled study on addictions (n=121) which was published in August 2005 in The American Journal of Drug and Alcohol Abuse, with David Kaiser, Ph.D, Siegfried Othmer, Ph.D, and Steven Sideroff, Ph.D. |
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Workshop #386): Protocol Efficiency
This workshop teaches practitioners how to recognize progress or the lack thereof in their EEG biofeedback clients. About one in three clients have extraordinary reactions to their first session but what about the others? It’s important for clinicians to be able to tell when people are responding in positive and negative ways. We’ll cover what types of things tend to improve first in the first few sessions when you have the right protocols. You’ll also learn what things tend to worsen when you have the wrong ones.
Workshop #387): Protocol Efficiency
This workshop teaches practitioners how to recognize progress or the lack thereof in their EEG biofeedback clients. About one in three clients have extraordinary reactions to their first session but what about the others? It’s important for clinicians to be able to tell when people are responding in positive and negative ways. We’ll cover what types of things tend to improve first in the first few sessions when you have the right protocols. You’ll also learn what things tend to worsen when you have the wrong ones.
Plenary #367): The Neurophysiology of Fear and State Specific EEG Changes
Fear states were induced in subjects to determine how their EEG, heart rate, and mental processing would change when compared to a neutral condition. We found significant differences in cognition, EEG and heart rate.
Subject’s (15-30Hz) amplitudes and cognitive functioning significantly dropped in amplitude while experiencing fear. Half of them had significant reductions in alpha (8-11Hz) and theta (5-8Hz). Doesn’t reducing alpha, theta, and high beta improve cognition? Or do we just need a new measuring stick for wave analysis that goes beyond amplitude, frequency and variability? Perhaps I’ve found one.
Details:
Workshop #386): Protocol Efficiency
We’ll spend the majority of this two hour course answering questions, going over cases and exploring the class’s experiences.
Workshop #387): Protocol Efficiency
If everyone had significantly abnormal EEGs we tend to see them regress to the mean. What about those who don’t. This is the majority of my practice and we’ll spend the majority of this two hour course answering questions, going over cases and exploring the class’s experiences.
Questions answered will be: How much beta? How much SMR? When do we start alpha-theta? What does it look like when people overdose on protocols? How do we handle overdoses? When do we need to add beta SMR to alpha theta protocols? How do you get a ballpark guess as to how many sessions one is likely to need? Why are negative reactions better than no reaction? How do we handle the placebo question? Why might we not be seeing beta or SMR significantly changing. What are common reasons people don’t respond to alpha-theta training? How do we help clients handle change they asked for yet didn’t expect to receive?
I’ll also cover language I use that seems to facilitate client understanding of the process. This makes it less uncomfortable to describe to their friends. I’ll also teach the class some techniques I’ve leaned on giving alpha-theta training for people with severe panic disorders.
Plenary #367): The Neurophysiology of Fear and State Specific EEG Changes
This talk is a follow-up to the talk Tom Brod and I shared last year entitled “What’s Happening When Nothing’s Happening?” If I’m able to gather enough data of this new metric from a new ADD study we’ve recently begun, you’ll hear it here first. It will explain why we don’t see people consistently declining in functioning when we reward for increases in alpha and theta. Or why can P300s or fMRIs show consistent increases and decreases functionality with beta-SMR training without changes in amplitudes in those bands. One thing we know for sure is that the brain isn’t amplitude modulated or we would make more noise going under power lines than our old AM radios. Or was it really the radio buzzing when we drove under them? I’m kidding {poke}.
Perhaps including this metric in the feedback process is part of the increased progress I’ve been having in the last year. It also could lead to new discoveries in other symptomatology.
Co-presenter with Thomas Brod of MYSTERIES OF NF SUCCESS: What's Happening When"Nothing's Happening"?
Contact Info:
Bill Scott
18219 Coastline Drive
Suite #1
Malibu, California 90265
phone: